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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle <t>lipidomics</t> in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.
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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle <t>lipidomics</t> in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.
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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle <t>lipidomics</t> in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.
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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle <t>lipidomics</t> in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.
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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle <t>lipidomics</t> in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.
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srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle lipidomics in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.

Journal: iScience

Article Title: Loss of SREBF1 disrupts lipid signaling and causes slow muscle fiber degeneration

doi: 10.1016/j.isci.2026.117447

Figure Lengend Snippet: srebf1 −/− ZF signaling networks converge to maintain slow muscle fiber (A) RNA-seq volcano plot showing differentially expressed genes in srebf1 KO vs. WT ZF skeletal muscle (FDR-significant genes highlighted in red and blue). (B) RNA-seq dataset identifies an srebf1 -centered transcriptional network connecting myogenic regulators ( pax3 , myf5 , myod1 , and myog ), slow-fiber–related structural genes (e.g., myh4 and tnnt1 ), and metabolic nodes ( pparg , ppara a , ppard , mlxipl , and mtor ). (C) Skeletal-muscle lipidomics in srebf1 KO vs. WT fish reveals altered endocannabinoids and PUFA-derived mediators (AEA, 2-AG, OEA, EPA, DHA, PGF2α, PGE2, PGD2, DiHOME species, and PEA) converging on nitric oxide, cAMP, TRPV1, cytokines (TNF and IL-6), Ca 2+ signaling, androgen receptor (AR), and “formation of myofibrils.” Together, these panels illustrate how srebf1 couples transcriptional programs of myogenesis with lipid-driven Ca 2+ -dependent pathways that support slow-fiber integrity.

Article Snippet: Lipidomics analysis , Shochat et al. , Shimadzu LabSolutions V5.65 software.

Techniques: RNA Sequencing, Derivative Assay

Journal: iScience

Article Title: Loss of SREBF1 disrupts lipid signaling and causes slow muscle fiber degeneration

doi: 10.1016/j.isci.2026.117447

Figure Lengend Snippet:

Article Snippet: Lipidomics analysis , Shochat et al. , Shimadzu LabSolutions V5.65 software.

Techniques: Cell-Signaling, Radio Immunoprecipitation, Lysis, Protease Inhibitor, Tissue, Microscopy, Knock-Out, Gene Knockout, Software, Micro-CT, Analysis